Division of Nephrology

Amanda Raff, M.D.

  1. Kopczynski, J., A. Raff, and J.J. Bonner (1992). Translational readthrough at nonsense mutations in the HSF1 gene of Saccharomyces cerevisiae. Molecular and General Genetics, 234: 369-378.
  2. Recht, J., Dunn, B., Raff, A., and M.A. Osley. (1996). Functional analysis of histone H2A and H2B in transcriptional repression in Saccharomyces cerevisiae. Molecular and Cellular Biology, 16(6): 2545-2553. 
  3. Spector, M., Raff, A., DeSilva, H., Lee, K., and M.A. Osley. (1997). Hir1p and Hir2p function as transcriptional corepressors to regulate histone gene transcription in Saccharomyces cerevisiae cell cycle. Molecular and Cellular Biology, 17(2): 545-552.
  4. Raff A., Susztak K., Schiffer M., and E. P. Bottinger (2004). Podocyte apoptosis as a mechanism of diabetic glomerulosclerosis. [Abstract – New York Society of Nephrology 2004]
  5. Susztak K., Raff A., Schiffer M., and E. P. Bottinger (2004). Podocyte apoptosis as a mechanism of diabetic glomerulosclerosis. [Abstract – ASN 2004]
  6. Raff A., Hebert T., Pullman J., and M. Coco (2005). Crescentic post-streptococcal glomerulonephritis with nephrotic syndrome in the adult: is aggressive therapy warranted? Clinical Nephrology, 63(5): 375-380.
  7. Schiffer M., Susztak K., Ranalletta M., Raff A., Bottinger E., and M. Charron (2005). Localization of the GLUT8 transporter in murine kidney and regulation in vivo in non-diabetic and diabetic conditions. Am J Physiol Renal Physiol, 289(1): F186-193.
  8. Kotlov M., Raff A., and V. Folkert (2005). A retrospective study of reticulocyte hemoglobin content as a means of assessing iron stores in hemodialysis patients with high inflammatory states. [Abstract – NKF 2005]
  9. Raff A., Hebert T., Pullman J., and M. Coco (2005). Crescentic post-streptococcal glomerulonephritis with nephrotic syndrome in the adult: is aggressive therapy warranted? Clinical Nephrology, 63(5): 375-380.
  10. Susztak K., Raff A., Schiffer M., and E. Bottinger (2006). Glucose – induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy. Diabetes. 55(1): 225-33.

Interim Division Chief

Vaughn Folkert, M.D (bio)

Professor of Clinical Medicine 

 
 

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Albert Einstein College of Medicine
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